AID for Immunoglobulin Diversity by Frederick W. Alt

By Frederick W. Alt

Advances in Immunology, a protracted tested and hugely revered serial, offers present advancements in addition to complete studies in immunology. Articles deal with the wide variety of issues that include immunology, together with molecular and mobile activation mechanisms, phylogeny and molecular evolution, and medical modalities. Edited and authored through the main scientists within the box, each one quantity offers up to date info and instructions for destiny learn.

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Begum, N. , and Honjo, T. (2006). Requirement of non‐canonical activity of uracil DNA glycosylase for class switch reccombination. J. Biol. Chem. 283, 732–742. , Pomerantz, R. , and Duan, L. X. (1998). Diversity of HIV‐1 Vpr interactions involves usage of the WXXF motif of host cell proteins. J. Biol. Chem. 273, 8009–8016. , Scharff, M. , and Goodman, M. F. (2003). Activation‐induced cytidine deaminase deaminates deoxycytidine on single‐stranded DNA but requires the action of RNase. Proc. Natl.

Lee, C. , and Honjo, T. (1998). Target specificity of immunoglobulin class switch recombination is not determined by nucleotide sequences of S regions. Immunity 9, 849–858. , and Bennett, S. E. (2005). Physical and functional interaction of human nuclear uracil‐DNA glycosylase with proliferating cell nuclear antigen. ) 4, 1421–1431. , and Maizels, N. (2001). DNA breaks in hypermutating immunoglobulin genes: Evidence for a break‐and‐repair pathway of somatic hypermutation. Genetics 158, 369–378.

Exp. Med. 197, 1291–1296. DiSanto, J. , Bonnefoy, J. , Gauchat, J. , and de Saint Basile, G. (1993). CD40 ligand mutations in x‐linked immunodeficiency with hyper‐IgM. Nature 361, 541–543. , and Honjo, T. (2003). De novo protein synthesis is required for the activation‐induced cytidine deaminase function in class‐switch recombination. Proc. Natl. Acad. Sci. USA 100, 2634–2638. Dudley, D. , Manis, J. , Zarrin, A. , and Alt, F. W. (2002). Internal IgH class switch region deletions are position‐independent and enhanced by AID expression.

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